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grk 5 in 2  (MedChemExpress)


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    MedChemExpress grk 5 in 2
    Grk 5 In 2, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/grk 5 in 2/product/MedChemExpress
    Average 94 stars, based on 5 article reviews
    grk 5 in 2 - by Bioz Stars, 2026-02
    94/100 stars

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    Figure 3—Adipose expression of Grk5 is a full mediator for RetroFat/BW (A) and EpiFat/BW (B) and a partial mediator for TRIG (C) at the Chr1 QTL. Panels A–C show mediation analysis for the phenotypes RetroFat/BW, EpiFat/BW, and TRIG, respectively. Within each row, left to right, are shown (i) box-whisker plots of the estimated founder haplotype effects with uncertainties at the peak SNP for the pheno- type; (ii) the scan across Chr1 for SNP-based mixed model for the trait; and (iii) the corresponding scan after including the expression of Grk5 as a covariate. Dash horizontal lines represent maximum log(P value) in physiological mapping of the traits. D shows the haplotype effects and expression QTL analysis for Grk5, in which the rightmost panel is a scatter plot of RetroFat/BW vs. Grk5 expression in adipose tissue across 415 rats, color-coded by genotype at the peak SNP of the pQTL for RetroFat/BW, showing the BUF and WKY allele. E shows that knockdown (KD) of Grk5 in a 3T3L1 preadipocyte cell line (left) decreases total TRIG accumulation in a mature adipocytes (right). The white bar represents the KD and the black bar is the control. D0, day 0; D7, day 7.

    Journal: Diabetes

    Article Title: Genetic Mapping of Multiple Traits Identifies Novel Genes for Adiposity, Lipids, and Insulin Secretory Capacity in Outbred Rats.

    doi: 10.2337/db22-0252

    Figure Lengend Snippet: Figure 3—Adipose expression of Grk5 is a full mediator for RetroFat/BW (A) and EpiFat/BW (B) and a partial mediator for TRIG (C) at the Chr1 QTL. Panels A–C show mediation analysis for the phenotypes RetroFat/BW, EpiFat/BW, and TRIG, respectively. Within each row, left to right, are shown (i) box-whisker plots of the estimated founder haplotype effects with uncertainties at the peak SNP for the pheno- type; (ii) the scan across Chr1 for SNP-based mixed model for the trait; and (iii) the corresponding scan after including the expression of Grk5 as a covariate. Dash horizontal lines represent maximum log(P value) in physiological mapping of the traits. D shows the haplotype effects and expression QTL analysis for Grk5, in which the rightmost panel is a scatter plot of RetroFat/BW vs. Grk5 expression in adipose tissue across 415 rats, color-coded by genotype at the peak SNP of the pQTL for RetroFat/BW, showing the BUF and WKY allele. E shows that knockdown (KD) of Grk5 in a 3T3L1 preadipocyte cell line (left) decreases total TRIG accumulation in a mature adipocytes (right). The white bar represents the KD and the black bar is the control. D0, day 0; D7, day 7.

    Article Snippet: The Grk5 gene was silenced by infecting 3T3-L1 preadipocytes with lentiviral particles to deliver mouse gene-specific shRNA expression vectors (sc-39043-V; Santa Cruz Biotechnology, Santa Cruz, CA) in the presence of polybrene (8 mg/mL; sc-134220; Santa Cruz Biotechnology) according to the manufacturer’s protocol.

    Techniques: Expressing, Whisker Assay, Knockdown, Control